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Research funded by RNID has made a major breakthrough towards using stem cells as a treatment for hearing loss in the future.
New research funded by RNID has successfully shown that loss of hearing is associated with spontaneous nerve activity within regions of the brain that process sound. This will help explain the causes of tinnitus, a vital step towards discovering a new treatment.
Following a detailed appraisal process, the National Institute of Health and Clinical Excellence (NICE) has announced guidance that paves the way for thousands of deaf adults and children who do not derive sufficient benefit from hearing aids to receive cochlear implants from the NHS. The news has been welcomed by a consortium of charities representing the UK's nine million deaf and hard of hearing people.
The study appearing in this month's Nature journal describes how a single gene could be used to induce the growth of inner ear hair cells, which are essential for hearing.
Smoking is a risk factor in the development of age-related hearing loss, one of the largest ever studies into risk factors for hearing loss has found.
A history of severe ear infections or tonsil trouble may increase the chances of being obese later in life, according to scientists.
Glutamate is thought to be the primary transmitter released by inner hair cells to activate spiral ganglion neurons in the auditory nerve. Signalling mediated by glutamate requires that this excitatory amino acid is loaded into secretory vesicles by a family of vesicular glutamate transporters (VGLUTs).
A gene linked to Otosclerosis, the single most common cause of hearing loss among white adults, has been identified.
RNID-funded scientists at the University of Antwerp have shown a genetic link between oxidation and noise-induced hearing loss, and in a separate study age-related hearing loss.
The Tinnitus Research Initiative (TRI) is a privately funded project dedicated to improving the quality of life for patients who have tinnitus.